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Nrf2 regulates oxidative stress and its role in cerebral ischemic stroke
Energy metabolic homeostasis can be disrupted in response to many risk factors
The CBBBP component could enhance the penetration of DSIP-CBBBP across BBB or improve its interactions with neural substrates involved in anxiety modulation
The result: telomerase catalytic subunit expression was reactivated, enzyme activity increased, and telomere lengthening occurred, effectively extending the cells proliferative capacity from ~34 to over 44 passages [1]
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